LangTurbo - Learn Languages Faster

Oké, dat ging niet helemaal goed.

Okay, that didn’t go quite right.

Ja, ik ga vragen. Jongens, hebben jullie Dr. Bikman nog kunnen horen?

Yes, I have questions. Guys, have you been able to hear Dr. Bikman?

Ik zag dat je verloor, maar ik denk dat ze nog steeds commenten maakten, dus ik heb dat idee van carbo-hydraten gewoon afgekomen.

I saw that you lost, but I think they were still making comments, so I just got rid of that idea of carbohydrates.

Ja, oké, laten we gaan.

Yes, okay, let's go.

Oké, maar ik wil niet dat iemand me verantwoordelijk maakt, want terwijl ik het vigorloos zeg en ik het verantwoordelijk maak,

Okay, but I don't want anyone to hold me accountable, because while I say it without vigor and I make it responsible,

zijn de mensen, de mensen die het verantwoordelijk maken, de mensen die het verantwoordelijk maken,

the people, the people who make it responsible, the people who make it responsible,

de mensen die het verantwoordelijk maken, de mensen die het verantwoordelijk maken,

the people who hold it responsible, the people who hold it responsible,

Some diseases are arising because of the insulin resistance itself, where the cell isn't responding very well to insulin.

Other diseases are happening because the body is swimming in a sea of insulin.

Other diseases are occurring because the body is swimming in a sea of insulin.

And that is hurting those cells that are still responsive to insulin.

Because some of the cells are still responsive and sensitive to insulin like they always were.

Omdat sommige van de cellen nog steeds responsief en gevoelig voor insuline zijn, zoals ze altijd waren.

But now there's just so much insulin that they're told to do too much.

Maar nu is er gewoon zoveel insuline dat ze te veel voorgeschreven krijgen.

And it works against the body.

Yeah, so then you also get stuff like high blood pressure.

Yeah, so then you also get things like high blood pressure.

That's also basically insulin resistance in the veins.

Dat is in wezen ook insulineresistentie in de aderen.

Am I correct?

Yeah.

So the cells change the lock.

Sugar is no longer welcome.

It stays in the body.

It glycates.

Is that correct?

Is dat correct?

Yeah.

And that will make the body just chronically inflamed.

And that will cause the body to be chronically inflamed.

Am I saying this right?

That's true.

Dat klopt.

Yeah.

So inflammation is a...

Is a very unique kind of player in this movie that we're talking about with insulin resistance and health.

Where inflammation can be a result of chronically elevated glucose.

When you are glycating amino acids in the blood, that starts to stimulate inflammation in the body.

When you glycate amino acids in the blood, it begins to stimulate inflammation in the body.

And in turn, inflammation is one of the key drivers of insulin resistance.

So if the body is experiencing higher levels of inflammation, that is a key drive or key component.

So if the body is experiencing higher levels of inflammation, that is a key driver or key component.

Because of insulin resistance.

Wegens insuline-resistentie.

Yeah.

And you also talk about waist circumference.

You know, if you have a bigger waist, that's also a very big indicator that you're insulin resistant.

So now the people are taking action.

They're dropping their carbs.

Ze laten hun koolhydraten vallen.

What is the role of ketones, which are basically not used when you're insulin resistant?

Only glucose.

What's happening with the ketones?

Once you drop the carbs and then you go, you know, you lower the inflammation.

And then the ketones come up and they are used as energy.

And then the ketones rise and they are used as energy.

Yes, you said it all very well.

So ketones have an actual caloric value comparable to glucose.

They have almost the exact same number of calories as glucose does.

They have almost the exact same number of calories as glucose.

So it's a very viable energy source.

The cells of the body are very hungrily using ketones.

The cells of the body are using ketones very eagerly.

Especially the brain.

Bijzonder de hersenen.

The moment ketones start going up in the blood.

The moment ketones start to rise in the blood.

The brain.

The brain starts using more ketones in place of glucose.

The brain starts using more ketones instead of glucose.

So in the normal individual who's eating lots of carbs all the time and they have high insulin.

The brain is using all glucose for energy and no ketones because if there's lots of insulin, there's no ketones.

The brain is using all glucose for energy and no ketones because if there's a lot of insulin, there are no ketones.

But the moment ketones start to go up, the brain will start using less glucose and start using more ketones.

But the moment ketones start to rise, the brain will begin to use less glucose and will start using more ketones.

So the brain perhaps prefers ketones as a fuel.

So the brain perhaps prefers ketones as fuel.

And that might be why people with migraine headaches and epilepsy and and Alzheimer's disease.

And that might be why people with migraine headaches, epilepsy, and Alzheimer's disease.

They start to feel better and think better when their ketones are higher.

It could be because you're fueling the brain a little better now.

But then to back up a ketone is essentially a piece of fat, a piece of burned fat.

And by that I mean when when insulin is low, the body really starts increasing its fat burning.

And by that I mean when insulin is low, the body really starts increasing its fat burning.

So the body has two primary fuels that are that are fueling most of our energy demands glucose and ketone.

So the body has two primary fuels that are fueling most of our energy demands: glucose and ketones.

In fact, glucose and fat.

In feite, glucose en vet.

And when insulin is high, the body is burning glucose and very little fat burning.

And when insulin is high, the body is burning glucose and very little fat.

When insulin is low, it shifts.

The body is burning very little glucose and very much, very high amounts of fat.

The body is burning very little glucose and a lot, very high amounts of fat.

And when insulin is low for an extended period of time, maybe about 20 hours or so, there's so much fat burning that the body is burning more fat than it needs to burn.

And that that extra fat burning is what starts to turn into ketones.

And that extra fat burning is what starts to turn into ketones.

All right.

So moving.

Zo ontroerend.

Yeah.

So if I'm right, it's also an energy problem with the brain, because when you're insulin resistant, the brain has the glucose, but it can't utilize it.

So it can't use it.

And that's basically the foundation of diabetes.

En dat is eigenlijk de basis van diabetes.

Is that correct?

Is dat correct?

Is that like the foundation of it?

Yeah.

Well, yes, very likely.

Yes.

The foundation of diabetes is that you can't you can't basically.

The foundation of diabetes is that you can't, you can't basically.

Well, there's lots of ways to maybe describe a foundation.

Well, there are many ways to perhaps describe a foundation.

But one of the problems is they they can't shift fuels anymore and they're constantly stuck trying to burn glucose.

Maar een van de problemen is dat ze de brandstoffen niet meer kunnen verschuiven en dat ze voortdurend vastzitten met het proberen glucose te verbranden.

But because the muscles have become insulin resistant and muscle is the main consumer of glucose and most of that.

Maar omdat de spieren insulineresistent zijn geworden en spieren de belangrijkste verbruiker van glucose zijn, en de meeste daarvan.

So muscle eats most of our glucose.

So muscle uses most of our glucose.

Eighty percent of the glucose we eat is going right into the muscle.

And so and then the rest of that is mostly going into fat, about 15 percent into fat cells.

And so then the rest of that is mostly going into fat, about 15 percent into fat cells.

But it needs insulin to open those glucose.

Maar het heeft insuline nodig om die glucose te openen.

Doors to the muscle cells.

And so the muscles becoming insulin resistant.

And so the muscles become insulin resistant.

Now, insulin is coming to the cells, the muscle cells and knocking on the door saying, hey, let the glucose in.

Now, insulin is coming to the cells, the muscle cells, and knocking on the door saying, hey, let the glucose in.

But the muscle doesn't open the door.

And so now the blood glucose levels climb.

And so now the blood glucose levels rise.

That's the foundation of type two diabetes.

Dat is de basis van type twee diabetes.

It's when the insulin resistance in the body has started to really affect the muscle.

Het is wanneer de insulineresistentie in het lichaam de spieren echt begint te beïnvloeden.

Now, glucose levels just skyrocket.

Now, glucose levels just soar.

Yeah.

So and a lot of I heard you talk about a lot of.

Some typical cancers are sensitive to glucose, like the breasts, the prostate and.

Some typical cancers are sensitive to glucose, such as those of the breast, the prostate, and.

There's another one.

Er is er nog een.

You have to help me.

Yes.

So breast and prostate cancers are the two that I always emphasize because they're the most common.

And both of those cancers are very often.

And both of those cancers are very common.

If you if you analyze the tumor cells, they have an overexpression of insulin receptors where they will have several times five.

If you analyze the tumor cells, they have an overexpression of insulin receptors where they will have several times five.

Or ten times the number of little doors for insulin to come and knock on.

And so you have these two most common cancers.

What's happening in the average person or the person who's insulin resistant is that they have a high amount of insulin in the blood.

And so that is stimulating the success of growth of these cancer cells because cancer cells, insulin tells cells to grow.

And the fact that the cancer cells have five to ten times more insulin.

Receptors means that there's five to ten times more responsive to this insulin signal.

Receptors means that there are five to ten times more responsive to this insulin signal.

So you have high insulin telling the cancer cells to grow rapidly.

And you have high glucose, which is the fuel for cancer cells.

Cancer cells use glucose for fuel.

Cancer cells gebruiken glucose als brandstof.

They don't use ketones.

They don't use fatty acids.

They use glucose.

And so we have this perfect storm.

High insulin fueling the telling or signaling the growth of the cancer cell.

High insulin fuels the telling or signaling of cancer cell growth.

High glucose fueling the growth of the cancer cell.

And now the cancer cells are just having a party, growing as rapidly as they want.

Wow.

Is that typical for every cancer?

Are these specific?

Yeah, I can't say.

That's a wonderful question.

Dat is een prachtige vraag.

There are more cancers than just breast and prostate.

But undoubtedly there are cancers that don't have an overexpression of insulin receptors.

Maar ongetwijfeld zijn er kankers die geen overexpressie van insuline-receptoren hebben.

But they still use glucose.

Maar ze gebruiken nog steeds glucose.

Jasper, I know of no exception to this.

Jasper, ik ken geen uitzondering op dit.

Every tumor in the body uses glucose as its fuel.

In fact, tumors love glucose so much.

And they use glucose, I think, 20 times or 200 times.

200 times more than any normal cell.

200 keer meer dan elke normale cel.

That when we actually want to see where tumors are in the body,

we will infuse what's called a radio-labeled glucose.

We zullen wat ze een radio-gelabelde glucose noemen toedienen.

It's basically a glucose molecule that has a very subtle radiation to it.

Het is in wezen een glucosedeeltje dat een zeer subtiele uitstraling heeft.

Not enough to hurt the body at all.

But it's enough.

Maar het is genoeg.

It's enough to detect with something called a PET scan.

Het is voldoende om te detecteren met iets dat een PET-scan wordt genoemd.

It's kind of like an MRI almost.

So we infuse the person with this radio-labeled glucose.

And then we measure their body.

And we can see where the glucose goes.

And these cancer cells, like if someone has a thyroid tumor,

And these cancer cells, as if someone has a thyroid tumor,

it pulls it in so fast that you see this thyroid that's just like glowing through the screen.

It pulls it in so fast that you see this thyroid that's just like glowing through the screen.

Because cancer cells use glucose as fuel.

Wow, Ben.

It actually makes me a little mad.

Because why aren't we screaming this in hospitals?

Waarom schreeuwen we dit niet in ziekenhuizen?

And why aren't we advocating this to big groups of people?

And why aren't we advocating this to large groups of people?

You know, I've been shot because of famous Instagram people in the Netherlands.

Because I tell these kind of things.

Omdat ik dit soort dingen vertel.

And they're like, that's crazy.

You know, that's crazy.

No, in fact, this is legitimate science.

No, in feite is dit legitieme wetenschap.

I am not saying a ketogenic diet should replace all chemotherapies and other cancer drugs.

No one is saying that.

Nobody is saying that.

But there is...

Maar er is...

There is work of a professor at Boston University.

There is work by a professor at Boston University.

He's a pioneer in this area.

His name is Thomas Seyfried.

S-E-Y-F-R-I-E-D, I think.

S-E-Y-F-R-I-E-D, denk ik.

I'm kind of glancing up at his book.

Ik kijk een beetje naar zijn boek.

I have it here somewhere.

Anyway, Thomas Seyfried at Boston University.

He finds that there are certain inoperable brain tumors, like a glioblastoma.

He finds that there are certain inoperable brain tumors, such as a glioblastoma.

This is a tumor that you cannot remove because you would just damage the brain and the person would die.

And putting people into ketogenic diets...

And putting people on ketogenic diets...

Totally solves the problem.

Totaal oplost het probleem.

It's exceptional.

So, I'm not saying a ketogenic diet or a low-carbohydrate diet should replace all other cancer therapies.

So, I'm not saying that a ketogenic diet or a low-carbohydrate diet should replace all other cancer therapies.

Not at all.

But, why not include it?

Maar, waarom het niet opnemen?

Because there is evidence to suggest a benefit.

Omdat er aanwijzingen zijn die een voordeel suggereren.

And my thought is, if I get cancer...

And I think about cancer.

My mom...

My mother died from breast cancer when we were...

My family, we were all quite young.

So, it's something I think about a fair amount.

So, it's something I think about quite a bit.

And I'm worried about it for my own sake.

And that's one of the reasons I generally adhere to a ketogenic diet.

Because I'm so afraid of cancer.

Omdat ik zo bang ben voor kanker.

Now, cancer is such a confusing, unknown origin.

So, I don't see a curing cancer in the very near future.

So, I don't see a cure for cancer in the very near future.

But even if the ketogenic diet can help me a little, then I'm going to go all in.

And so, my advice to anyone is, yes, listen to your doctor.

Explore the best available therapies.

Explore de beste beschikbare therapieën.

But I would definitely...

Maar ik zou zeker...

If I were talking to a loved one, I would say, it's time to go ketogenic all the way.

Yeah.

My mother actually had breast cancer last year.

So, she went on a ketogenic diet and lost a fair amount of weight.

So, I'm really happy about that.

And I'm also scared...

And she's doing well now?

Yeah.

Her medication...

Actually, my next question is, her medication...

They took away two medications.

And a medication is basically like insulin, right?

Because they bump up the insulin.

Omdat ze de insuline verhogen.

Insulin is higher when you're diabetic with the medicine.

I saw that in your video and I was shocked.

And I couldn't sleep anymore.

Like, is this real life?

What is this?

It's crazy.

And Jasper, we know, we have data that when you treat...

So, this is...

I don't mean to get off on too much of a tangent.

I don't mean to go off on too much of a tangent.

But in type 2 diabetes, insulin is high.

Maar bij type 2 diabetes is insuline hoog.

It's just that it's not working well.

But the traditional view is, well, we have high insulin.

But we also have high glucose.

Maar we hebben ook hoge glucose.

So, let's just push the insulin up even higher by giving them injections.

And now that will lower the glucose.

It does lower the glucose.

But they are two times more likely to die from cancer when you do that.

If you give a diabetic insulin, you double their risk of dying from cancer.

And you triple their risk of dying from heart disease.

So, when we give a type 2 diabetic insulin, not type 1, but type 2,

So, when we give insulin to a type 2 diabetic, not type 1, but type 2,

we make them fatter and we kill them faster.

But it's a wonderful way to sell more insulin.

Maar het is een geweldige manier om meer insuline te verkopen.

But why are they using it?

Maar waarom gebruiken ze het?

What's the reason behind this medicine?

What's the reason?

I honestly think it's because maybe two reasons.

I honestly think it's because of maybe two reasons.

One, they don't understand how dangerous high insulin is.

That might be a true ignorance on their part.

Dat zou een ware onwetendheid van hun kant kunnen zijn.

And then cynically, number two, it might just be that they want to make a lot of money.

And I don't like to speak evil of drug companies in general.

And I don't like to speak ill of drug companies in general.

Because I think it is a miracle of science that we have the drugs that we do.

Omdat ik het een wonder van de wetenschap vind dat we de medicijnen hebben die we hebben.

But in the case of insulin in type 2 diabetes, I think that is just a way to make a lot of money.

Maar in het geval van insuline bij type 2 diabetes, denk ik dat het gewoon een manier is om veel geld te verdienen.

That insulin should be for type 1 diabetes.

In type 2, you just, you know, I won't say you never need it, but it would be very, very rare.

Wow, this is, it's basically shocking to hear this live.

You know, I've seen the videos, but to hear it again and to know that basically the insulin medication is making diabetes,

You know, I've seen the videos, but to hear it again and to know that basically the insulin medication is causing diabetes,

people with diabetes,

more sick and die faster, that's crazy.

You just want to scream to people, you know, stop eating sugar and it's going to be better.

You just want to scream at people, you know, stop eating sugar and it will get better.

Because is it, can you completely heal diabetes?

Because is it possible to completely heal diabetes?

Can you completely get rid of it?

Yeah, so in type 2 diabetes, yes, yes.

Now Jasper, even that is kind of clever, the way people will talk about it.

Because conventional thinking, like in a clinic, they will say type 2 diabetes is irreversible.

Because conventional thinking, like in a clinic, will say that type 2 diabetes is irreversible.

It is incurable.

Because if you follow their advice, it is incurable.

Want als je hun advies opvolgt, is het ongeneeslijk.

So imagine someone who comes to the clinic, they just get diagnosed with type 2 diabetes.

So imagine someone who comes to the clinic, they just got diagnosed with type 2 diabetes.

And the physician or the nurse is saying, okay, here is your prescription for some medications.

Rip it off and go take this to the pharmacy.

Tear it off and go take this to the pharmacy.

If they try to treat their type 2 diabetes with medications, it will never go away.

They will always need to take medications.

It will never cure the problem.

And so that's why they will say that.

Because if you follow their typical strategy, which is taking drugs, then it will never get better.

Because diabetes, type 2 diabetes is not a disease that is caused because you aren't taking drugs.

Because diabetes, type 2 diabetes is not a disease that is caused by not taking medication.

It's a disease of your diet.

And so if that conversation in the clinic goes, rather than, okay, here is your prescription for a drug.

If they say, here is your prescription to change your diet.

Go meet with this dietician.

Go meet with this dietitian.

And the dietician knows that this is a disease of too much glucose and too much insulin.

And so let's just lower your glucose.

Let's lower your insulin.

Laten we je insuline verlagen.

And now the disease starts to get better.

Because that is the truth.

Omdat dat de waarheid is.

Type 2 diabetes is a dietary disease.

And only when we change the diet do we actually improve the disease.

But this sounds crazy to some people.

Maar dit klinkt gek voor sommige mensen.

Other people, they say, that makes all sense.

Other people, they say, that makes complete sense.

all the sense in the world. That is perfectly

rational. I even like

to end that thought by mentioning

to conclude that thought by mentioning

some diabetes drugs. So there are

some of the popular diabetes

drugs are called

SGLT inhibitors.

SGLT

inhibitors, they're two different types.

Inhibitors, there are two different types.

They will try to

block the digestion of glucose from our

diet. So we eat a bagel and

you take an SGLT inhibitor and

you just don't move that glucose into

your blood and it stays in your guts.

And it gives you terrible diarrhea

And it gives you terrible diarrhea.

unfortunately. But there's

Unfortunately. But there's

another type which you take this

een ander type dat je dit neemt

drug and then it basically starts pushing

drug en dan begint het in wezen te duwen

all your glucose from your blood

into your urine so that you urinate

it out. Then you have a much, much higher amount

of urine. But in that case

you're putting so much glucose in your urinary

you're putting so much glucose in your urine

tract that they're much more likely to

have infections because there's always

bacteria in the urinary tract

and bacteria like cancer cells

use glucose as their fuel.

So we basically are just giving the bacteria

So we basically are just giving the bacteria.

all the sugar they want to grow and so they grow

very rapidly. But what's so crazy

about this, we're giving a person

a drug to block their glucose

digestion in their guts

or we're giving them a drug to push the

glucose out into their urine

and then I'm just standing on the side

whispering, why not just eat

less glucose? Let's just eat

less glucose. If that's what you're trying to push

out of the body, then just put less in the body

in the first place. And that's

in the first place. And that’s

why a low carbohydrate diet

can in fact,

cure type 2 diabetes. You can put

someone with type 2 diabetes on a low

carbohydrate diet, they can get off

every single medication and have

no clinical

signs of the disease anymore.

Wow, this is amazing.

Wow, dit is geweldig.

This is really good for the people in

Europe and Holland and Belgium

Europe and the Netherlands and Belgium

and also some people from

en ook sommige mensen van

the United States here. So Ben, I'm looking

at the time. Can I ask one more question?

Just a final question about

the ketogenic diet. It's

a very important

debate we had with

a lot of people from Instagram that are

promoting this high carb, low fat diet

and counting

en tellen

calories. That's

really something, if you could elaborate

Really something, if you could elaborate.

on counting calories is not an

Counting calories is not a

efficient way to lose weight and get healthy,

that would be amazing.

That would be amazing.

Y'all are happy to.

Jullie zijn er blij mee.

So I think counting calories is

a short term way

a short-term way

to lose weight,

but it's not viable long term.

maar het is op lange termijn niet levensvatbaar.

And there's a lot of reasons

And there are a lot of reasons.

for this, but maybe I'll take a step

for this, but maybe I'll take a step back

back and present my big view.

My view on

obesity and fat

obesitas en vet

cells and the growing and shrinking of the

fat cells is that you can

Fat cells is that you can

shrink fat cells

through two different mechanisms.

door twee verschillende mechanismen.

One is controlling

One is controlling.

the energy. In other words, low calorie.

You just start robbing the

body or depriving it of energy.

Yes, it's going to start using

more of its own energy and then

body weight will go down, body fat will go down.

Body weight will decrease, body fat will decrease.

Also, there's

low insulin.

The low insulin strategy

acknowledges that insulin

totally controls the growing

and the shrinking of a fat cell.

That is 100% truth.

That is 100% true.

There's no getting around that fact.

Daarom is er geen ontkomen aan die waarheid.

You literally, as we

grow fat cells in my own lab,

right across the hallway, you cannot

recht tegenover de gang, kun je niet

make a fat cell grow unless

insulin is high. In contrast,

you cannot make a fat cell

you cannot create a fat cell

shrink unless insulin is

low. It is impossible.

laag. Het is onmogelijk.

Now, I'm not saying

energy doesn't matter.

Energy doesn't matter.

I'm not saying calorie number doesn't matter.

I'm not saying the calorie number doesn't matter.

But, there's something really clever

Maar, er is iets echt slim.

that the body can do when insulin

what the body can do when insulin

changes. When insulin goes down,

veranderingen. Wanneer insuline daalt,

metabolic rate

goes up, and

ketone production goes up. And I'll mention

that point in a moment, but when

dat moment, maar wanneer

people are trying to go into

perfect thermodynamic balance

with calories in, calories out,

you can never know.

Exactly how much calorie you're putting

Exactly how many calories you're putting in.

in. You can never,

ever know exactly how much

ever weten precies hoeveel

calorie is going out.

Calorie is being burned.

It's impossible. That's part of the problem

It's impossible. That's part of the problem.

with the calorie in, calorie out. You cannot

know the truth.

The true

fidelity or accuracy

of trying to count calories. But,

or trying to count calories. But,

when insulin is low,

we are burning about 300

calories more per day

than when insulin is high. That's a lot

of excess calorie burning.

And, if you're making ketones,

En, als je ketonen aanmaakt,

remember, a ketone is a piece

Remember, a ketone is a piece.

of fat. And this is,

and when you have higher ketones,

you are breathing them out,

and you are urinating them out.

That is actual caloric energy

That is actual caloric energy.

that we didn't have to store,

that we didn't have to work hard

and work out to burn. We just wasted

it from the body. So, we have

it uit het lichaam. Dus, we hebben

this kind of third way of

accounting for calories

that no one ever actually accounts

for. And so, my view on

obesity is, yes, calories matter.

Obesity is, yes, calories matter.

But, if we overlook what

Maar als we over het hoofd zien wat

those calories do to insulin,

then we're missing what I consider

the most important piece of the puzzle.

Because, if insulin is high, metabolic

rate will slow down, and the body wants to

the rate will slow down, and the body wants to

store energy more. If insulin is

store more energy. If insulin is

up, sorry, if insulin is low,

then metabolic rate goes up, and fat cells

are breaking down fat, and the body is

using energy, and metabolic rate, of course,

is high. Now,

having said all of this, one

Having said all of this, one

last comment. My

frustration with the low-calorie approach

frustratie met de caloriearme benadering

is that it is based on hunger.

And hunger

always wins. This

is why I say there's a short-term

benefit, but hunger

voordeel, maar honger

always wins, which is why if you're

altijd wint, daarom als je bent

calorie counting, you almost always will lose,

calorie counting, you will almost always lose,

and you'll go right off it and gain all

and you'll go straight for it and gain everything.

your weight back. But, if you start

je gewicht terug. Maar als je begint

your weight loss journey

with a low-insulin approach,

that isn't based on eating less.

The low-insulin approach just

says control your carbohydrates,

you know, focus more on fruits and vegetables,

You know, focus more on fruits and vegetables.

very little grains, very, very

little sugar, and then

focus more on protein and fat.

And you can eat when you're hungry,

and when you're not hungry, don't eat.

That's really the key, so you don't have to

Dat is echt de sleutel, zodat je het niet hoeft te.

be hungry. And that's

the low-insulin approach. So my idea,

your journey is, start with the low-insulin

Your journey is to start with the low-insulin.

approach. Control carbohydrates,

prioritize protein, don't fear fat.

And then, you will

En dan, zul je

lose weight, and then you'll get to

a plateau, usually.

And if you get to where you stop

En als je bij de plek komt waar je stopt

losing weight, but you want to lose more,

now focus on a

lower-energy approach. Now

maybe start fasting more

often. You know, before you were

fasting just because you weren't hungry,

now we say, fast

because you're planning it. You have

omdat je het plant. Je hebt

a structured fast, and now you're

lowering your energy coming in, and

now the low-insulin and the low-energy

means you'll go from that first weight-loss

means you'll go from that first weight loss

plateau to the next weight-loss plateau,

and now you've done it best. So,

we insulin advocates,

me and you, we're not

saying calories don't matter.

We're not that foolish, and we're not that ignorant.

We’re not that foolish, and we’re not that ignorant.

We're just saying

We zeggen het gewoon.

hormones also matter, and maybe

they matter more in a long-term

ze tellen meer op de lange termijn

view. And maybe one last thought,

Jasper, the more someone goes on

a low-fat diet, the more they're

eating foods that they actually don't

need. Because we don't need

carbohydrates in our diet, we

need fat, and we need protein.

We need fat, and we need protein.

Literally, those are

necessary for human survival.

That's what we should be focusing on.

Daar zouden we ons op moeten richten.

Wow, Benjamin.

It's like a mic drop right now.

Het is nu als een mic drop.

You know, you said it all. And it's also

to end with that, a little bit

to finish with that, a little bit

of the satiety hormones.

You know, the ketones

You know, the ketones.

make sure we are not

as hungry as when insulin is

high. So, am I correct? Right.

High. So, am I correct? Right.

That's correct, right? Yeah.

Dat klopt, toch? Ja.

So, we should focus on

that. It's, wow.

dat. Het is, wauw.

It's mind-boggling, and I'm

a little bit scared,

you know, because of the

doctors basically not helping

us in the right direction. They should

basically say, no medicine, let's try,

let's first try a low

carbohydrate or ketogenic diet

and then look if maybe medicine

and then see if maybe medicine

is justifiable, but

is gerechtvaardigd, maar

that's down the road.

dat is verderop de weg.

Yeah, no, and Jasper, one,

and this is my compliment to you,

we need more people like you.

We need more people like you.

Because

if the person can't get this in the clinic,

and you and I aren't speaking evil

of all doctors, we aren't, because

you only know what you've been taught.

You only know what you've been taught.

And this just isn't something that is

En dit is gewoon niet iets dat is

taught in medical schools. And so

there are those of us that have

kind of looked through the data, and that's

I kind of looked through the data, and that's

my job as a scientist, literally,

and seen, wait a minute,

we were a little wrong on some of these things, and there's

We were a little wrong about some of these things, and there's

another view that might be better

een ander uitzicht dat misschien beter is

and more accurate.

en nauwkeuriger.

If we can't convey that to a patient

in a clinic, all the more reason to

convey that on something like social media

directly to the person, with the

hopes that this person

can make the changes because

of their own studies,

their own research. And they don't have to wait

their own research. And they don't have to wait.

for a clinical visit to be told

what to do.

Yeah, that's amazing. And I will keep going

Yeah, dat is geweldig. En ik zal doorgaan.

and tell the message in the Netherlands,

Europe, Belgium, all

the people that are willing to listen

to me, and

I will tell them, buy the book,

I will tell them, buy the book.

read the book, it's life

changing. It changed my life,

and also my mother's life

en ook het leven van mijn moeder

who had breast cancer,

she's now on a low carbohydrate

she's now on a low carbohydrate diet

diet, and she's doing great.

dieet, en het gaat geweldig met haar.

So it's a life,

really it's a life-saving book. So Ben,

you made my day. I was a bit nervous,

but I want to thank you,

I want to thank you so much for this conversation.

I'm really happy

Ik ben echt gelukkig.

how it all went, and I just want

to thank you. Thank you so much.

My pleasure, my pleasure. Let's keep in touch.

Happy to be a resource, and

Blij om een bron te zijn, en

truly grateful to be

kind of on the front lines with you, or part of the same

army, fighting the same battle,

to just give knowledge to

to simply provide knowledge to

people, and then let them, with all of the

knowledge, hiding nothing, let them make

their own decisions. Great.

hun eigen beslissingen. Geweldig.

Thank you, Ben. I want to wish you a great

day, and I will talk to

you maybe sometime later.

maybe you sometime later.

Thank you. Thanks, Jaster.

Bye, everyone. Bye-bye.

In gesprek met Dr. Benjamin Bikman over insulineresistentie